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Figure 5–6. The day-of-injury (DOI) computed tomography (CT) scans from Figures 5–4 and 5–5 have been analyzed in 3-D space outlining the ventricle (aquamarine color) on the DOI (A, left) compared with the follow-up magnetic

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Figure 5–6. The day-of-injury (DOI) computed tomography (CT) scans from Figures 5–4 and 5–5 have been analyzed in 3-D space outlining the ventricle (aquamarine color) on the DOI (A, left) compared with the follow-up magnetic resonance imaging (MRI) on the right (B), performed 2 years postinjury.Note the obvious ventricular dilation, a sign of nonspecific parenchyma volume loss. The bottom left CT scan (C) shows the multiple hemorrhagic lesions in red as occurred on the DOI, identified by points of increased density as shown in Figures 5–4 and 5–5. Note their congregation in the frontotemporal and periventricular regions. Superimposed on the 3-D follow-up MRI (D) is the combination of white matter signal changes (shown in red) revealed on the fluid-attenuated inversion recovery sequence and the regions of hemosiderin deposition identified in the gradient recalled echo sequence (shown in yellow).

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