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Published Online: March 1946

COMPLICATIONS IN ELECTRIC SHOCK THERAPY

Publication: American Journal of Psychiatry

Abstract

Therapeutic electric shocks produce some reversible cortical changes, probably together with some irreversible neuronal degeneration and gliosis. The typical memory losses are generally recoverable, and the diversified EEG disturbances tend to disappear in several months.
According to reports in the literature, the neuronal discharge may have other effects than the intended grand mal: cardiac arrest, autonomic disorders, status epilepticus, or manic delirium.
Regardless of operating technique, reported rate of compression fracture of a vertebral body vary from 0.5 percent of cases, to 20 percent with routine X-raying. Many compressions will remain undiscovered unless spines are routinely X-rayed post-shock. Compression spinal fractures are clinically inconsequential. The humerus, or more rarely some other bony structure, is occasionally fractured; to these instances the technique of shocking seems relevant.
Dislocation at the shoulder or mandibular joint should be technically preventable.
Arterial hypertension may be aggravated by electroshock, and myocardial insufficiencies can lead to a fatal outcome. Curare attenuates the convulsive violence but may add new dangers; its drawbacks are still under scrutiny.
Aspiration during the coma has been deemed responsible for complicating lung abscesses. Liability to pulmonary complications probably has other unknown causes. Post-shock pneumonias have not always been charged to the therapy. The published mortality rates appear over-optimistic.

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Published In

Go to American Journal of Psychiatry
Go to American Journal of Psychiatry
American Journal of Psychiatry
Pages: 594 - 598
PubMed: 21026841

History

Published in print: March 1946
Published online: 1 April 2006

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LOUIS LOWINGER
Neuropsychiatric Research Unit, Veterans Facility, Northport, L. I., N. Y.
JAMES H. HUDDLESON
Neuropsychiatric Research Unit, Veterans Facility, Northport, L. I., N. Y.

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