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Letter to the Editor
Published Online: 1 November 2000

New DSM-IV Diagnosis of Acute Stress Disorder

To the Editor: I applaud Dr. Marshall et al. for their analysis of the validity and utility of the acute stress disorder symptom criteria and diagnosis; however, I do not draw the same conclusions from the findings reported to date. The fact that acute stress disorder falls short in predicting PTSD does not necessarily undermine its validity; many trauma victims show natural recovery over time (1), and some sufferers experience a delayed onset of symptoms. Moreover, the difficulty in differentiating normative and pathological posttraumatic reactions in the immediate aftermath of an event highlights the failure to identify the elements of the process that underlie the pathology, elements that apparently are not fully captured by the symptoms of either acute stress disorder or PTSD. In addition, findings regarding the predictive power of a variety of peritraumatic reactions (2), including dissociation, and reports of the ubiquity of dissociative symptoms in posttraumatic conditions (Butler et al., 1996) indicate that broadening our conceptions, rather than limiting them, may be most useful. Indeed, subtyping reactions (into, for example, types that principally involve dissociative versus hyperarousal or anxiety symptoms) may have utility. Individual differences, event characteristics, and features of the recovery environment (3) may also differentiate symptom profiles and courses. For example, individual differences in the facility or propensity to dissociate may represent a diathesis for the development of longer-term dissociative conditions, including PTSD, under conditions of extreme stress (Butler et al., 1996). The authors’ assertion that the two diagnoses cleave essentially continuous clinical phenomena actually begs the question.
Consequently, I believe that the findings suggest that the time has come for an extensive empirical investigation into the constituents of peritraumatic, acute, and longer-term posttraumatic reactions, including predictors of chronicity. By thoroughly documenting the elements of these reactions—without the Procrustean constraints of the current acute stress disorder and PTSD diagnoses—we will be able to construct empirically defensible diagnoses that truly fit the clinical phenomena.

References

1.
Kessler RC, Sonnega A, Bromet E, Hughes M, Nelson CB: Posttraumatic stress disorder in the National Comorbidity Survey. Arch Gen Psychiatry 1995; 52:1048–1059
2.
Bernat JA, Ronfeldt HM, Calhoun KS, Arias I: Prevalence of traumatic events and peritraumatic predictors of posttraumatic stress symptoms in a nonclinical sample of college students. J Trauma Stress 1998; 11:645–664
3.
Green BL: Identifying survivors at risk, in International Handbook of Traumatic Stress Syndromes. Edited by Wilson JP, Raphael B. New York, Plenum Press, 1993, pp 135–144

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Go to American Journal of Psychiatry
Go to American Journal of Psychiatry
American Journal of Psychiatry
Pages: 1884-c - 1884

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Published online: 1 November 2000
Published in print: November 2000

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LISA D. BUTLER, PH.D.
Stanford, Calif.

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