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Disorders of mood and affect are common consequences of traumatic brain injury (TBI). The pathogenesis of these disorders following TBI is complex and, in all likelihood, variable with respect to etiology. In some cases, especially in the early period following TBI, the development of these disorders may reflect the effects of neurotrauma on the several distributed neural networks that generate and regulate emotion (Jorge et al. 1993a). For some patients, particularly those in whom depressive disorders develop in the late postinjury period, psychological and social factors appear to be etiologically important (Jorge et al. 1993a; Whelan-Goodinson et al. 2010).
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